Furthermore, lysis of host cells by CDCs helps bacteria avoid the bactericidal activity of phagocytic cells and aids in evasion of the host immune response. About Search query Search Twitter. Murine model of pulmonary anthrax: kinetics of dissemination, histopathology, and mouse strain susceptibility. The lethality of rALO and native ALO on human monocytes, neutrophils, macrophages and lymphocytes supports the idea that ALO may represent a previously unidentified virulence factor of B. Find what's happening See the latest conversations about any topic instantly. We hypothesize that ALO can be defined as a new B. Recombinant ALO lyses primary human lymphocytes in a dose dependent manner. The means and standard deviations of three independent experiments performed in duplicate are shown. These epithelial cells represent those that B. In all forms of anthrax, gastrointestinal, pulmonary, or cutaneous, B.
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An Arcanobacterium Actinomyces pyogenes mutant deficient in production of the pore-forming cytolysin pyolysin has reduced virulence. Back Next.
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The presence of free cholesterol inhibits ALO-mediated lysis [ 8 ]. Streptococcus suis interactions with the murine macrophage cell line J adhesion and cytotoxicity.
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Hemolysis assays revealed that the parent strain,grown under the same conditions, produces approximately 1,fold less ALO.
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Learn more. Figure 2. Murine model of pulmonary anthrax: kinetics of dissemination, histopathology, and mouse strain susceptibility. Viability was measured by trypan blue exclusion and verified using an LDH based cytotoxicity assay.
Sensitivities of human monocytes and epithelial cells to pneumolysin are different.
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|Percent viable THP-1s in the presence of B.
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The means and standard deviations of three independent experiments performed in duplicate are shown. The biology of CSF-1 and its receptor.
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